Inicio

    Se encuentra usted aquí

    • You are here:
      • Inicio > Molecular analysis of the non-cell autonomous effects in Down syndrome cortex using mouse ESC-derived brain organoids

Molecular analysis of the non-cell autonomous effects in Down syndrome cortex using mouse ESC-derived brain organoids

Molecular analysis of the non-cell autonomous effects in Down syndrome cortex using mouse ESC-derived brain organoidsMolecular analysis of the non-cell autonomous effects in Down syndrome cortex using mouse ESC-derived brain organoids

Summary

Down syndrome (DS) is the most frequent genetic cause of intellectual disability due to trisomy of human chromosome 21. In the brain, the pathological sequelae of trisomy 21 results in intellectual disability in infants as well as early onset dementia in adults. 

Start: 20/04/2020 End: 18/01/2023